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Rly regulated, overexuberant response to infection so that levels of expression after infection often exceed those seen in WT mice. It is also of interest to note that several of the proteins (13 proteins out of 59) that were present in increased amounts in the uninfected SP-A-/- mice respond to infection by reducing their levelsAli et al. Proteome Science 2010, 8:34 http://www.proteomesci.com/con
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Riments performed in duplicate and normalized to the amplification of ACTB mRNA. Bars represent mean ?SEM of the fold change with respect to untreated. *p
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Tion, and additional tumor initiating pathways promote mammary carcinogenesis. We have previously shown that activated ERK1/2 levels and the migratory action of TERT-siSFRP1 cells are drastically reduced in response to TGF-R inhibition which is consistent with work described by Imamichi et al. showing that TGF- signaling mediates the cellular migration of breast cancer cells by ERK1/2 activation [
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Tend to be more difficult and expensive to store and toTend to be more difficult and expensive to store and to distribute, since viability must be maintained, often requiring formulation approaches [7] for stabilization . On the other hand, killed/inactivated vaccines have a number of disadvantages. The major challenge is that since cells are never infected with the live microbe, these vacci
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An N-terminus with small acidic motif (DSpSpEE) that is also found in SIBLING proteins [65]. When solubilized, statherin has a random coil conformation. When bound to HA, it takes on a more alpha-helical structure exposing a bacterial binding site [63], leading to bacterial entrapment. Another example is the role of the IDP, DMP1, in preventing kidney and cardiovascular calcification. The double k
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